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Studies from various organisms implicate DNA replication factors in the inheritance of heterochromatin silencing.
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The production and the phenotypic analysis of pairs of nearly isogenic lines (NILs) differing only for the allele constitution at given chromosome regions provides the opportunity to test for the presence at such regions of genetic factors involved in the inheritance of a quantitative trait [ 1, 2].
There are other complex pathways for the transmission of a phenotype across generations without the transmission of a specific common or rare variant, namely through epigenetic factors that can result in the inheritance of gene expression patterns without an alteration of the DNA sequence [ 20].
This analysis indicates some important factors linked to the inheritance of pyrethroid resistance trait.
The best known genetic risk factor is the inheritance of the ε4 allele of the apolipoprotein E (APOE).
It is not often realized that Darwin believed in the inheritance of acquired habits.
This is referred to as epigenetic transgenerational inheritance [ 5, 15] and suggests a role for environmental epigenetics in the inheritance of phenotypic variation and disease, independent of classic genetic inheritance mechanisms.
Therefore, a relatively low number of haplotypes is sufficient to describe the inheritance of these factors for protein yield.
Alternatively, the inheritance of common genetic factors for cardiac defects and MA has been suggested.
In a later study in Poa, a five locus model for the inheritance of apomixis confirmed the independent inheritance of apospory and parthenogenesis [ 35].
The inheritance of resistance in G978 was studied using M4 and BC1F2 segregation data.
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