Sentence examples for factor mobilization from inspiring English sources

In addition, the temporal relationships of host tissue ingrowth, bioactive factor mobilization, scaffold degradation and immune cell infiltration, as well as the reciprocal signaling between discrete cell types and scaffolds are discussed.

The aim of this study was to examine the spontaneous in vivo recolonization potential of xenogeneic decellularized heart valves in a lamb model and the effects of granulocyte colony-stimulating factor mobilization of bone marrow cells on this process.

It is carefully controlled by a large number of secreted factors that bind to receptors on endothelial cells, as well as negative regulators that inhibit angiogenesis through direct effects on endothelial cells or indirect effects on growth factor mobilization and activation [24], [37].

About 0.01 to 0.004% of mononuclear cells from peripheral blood donors, who had received granulocyte colony-stimulating factor mobilization for transplant purposes, contained the viral genome [ 32].

These drawbacks can be overcome by combining G-CSF with other cytokines and using different growth factor mobilization strategies [ 18– 20].

Thanks to their proteolytic activity, MMP-9 and MMP-2 promote ECM-stored growth factors mobilization, including VEGF, thus favoring angiogenesis in HCC [ 120, 121].

23– 26 This collection failure results in increase resource utilization in terms of marked increase in use of growth factors, mobilization reattempts using chemotherapy, hospitalizations, need for transfusions, and antibiotics for neutropenic fever.

Activation of PARs upregulates endothelial expression of tissue factor (TF), mobilization of Weibel Palade bodies and release of von Willebrand factor [ 10, 11].

Moreover, stimulation of epidermal growth factor causes mobilization of α6β4 integrin from the hemidesmosomes and increases formation of lamellipodia and membrane ruffles that contain α6β4 integrin [ 18, 19].

Although underlying disease has been reported as a factor influencing mobilization, GITMO-WG deemed that this is not an independent factor, being influenced by the previous treatment and by the disease status.

Mammalian CXCR4 forms with TNFalpha, several HSP proteins and GDF5 an activation cluster involved in monocyte LPS signal transduction [ 57] and also acts synergistically with the colony-stimulating factor mediated mobilization of hematopoietic stem cells [ 57, 58].