Many studies have found continued expression of E6 and E7 proteins in the majority of cervical cancer cases, but not in normal tissues.
The mechanism for EGFR overexpression in the majority of cervical squamous cell carcinomas remains to be identified.
Human papillomavirus (HPV) types 16 and/or 18 are implicated in the majority of cervical cancers globally and local data would suggest these types are responsible for approximately 80% of cervical cancers in Scotland (Cuschieri et al, 2010).
In these experiments we detected over-expression of the Δ2 variant in the majority of cervical SCC shown in previous analyses to over-express p73α.
In countries with an organised gynaecological screening, the majority of cervical cancer cases occur among women who have not attended midwife surgeries for collection of a cervical smear (Andrae et al, 2008).
Patterns of cervical cancer differ between developed and developing countries, with the majority of cervical cancer burden occurring in developing countries, typically detected via symptoms at later invasive stages [11].
Due to widespread screening programs, the majority of cervical cancer cases are being diagnosed in the earlier stages.
Persistent infection with Human Papillomavirus (HPV) type 16 causes the majority of cervical cancers.
HPV is one of the most common sexually transmitted infections and causes the majority of cervical cancers.
The majority of cervical cancers (52%) were E-cadherin positive, with a complete absence of the antigen in only 10% of the tumours.
