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To evaluate the potential role of oxidative stress in the evolution of cervical cancer, including its pre-malignant states.
Hence, the expression of these two chemokine axes is likely to be tightly linked in the evolution of cervical cancer.
Conclusions: The most important prognostic factor in the evolution of patients with a cervical carcinoma is the stromal invasion depth followed by tumour size.
In summary, our findings suggest that the chemokine axes CXCL12/CXCR4 and CXCL16/CXCR6 are tightly linked and may form a network to mediate the evolution of cervical cancer.
This article reviews the evolution of cervical interbody implant designs and assesses future research directions.
It is well-established that persistent infections caused by human papillomavirus (HPV) is a key aetiological factor in the development of cervical lesions and cervical cancer [ 3].
Persistent HPV infection is the central factor in the development of cervical cancer, and is a prerequisite for progression to high-grade cervical lesions [ 15].
Given the high incidence of cervical dysplasia [ 15] and that cervical cancer rates remain high at 27.5/100,000 [ 16], we looked at whether HC use in the population was a factor in the development of cervical dysplasia and cancer.
Polymorphism of the p53 gene, codon 72, is also considered a risk factor in the development of cervical carcinoma.
Persistent infection with high risk Human papillomavirus (HPV) is the main aetiological factor in the development of cervical cancer.
Other bacteria, Chlamydia trachomatis (Ct), infection acts as a risk factor in the development of cervical lesions.
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