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Thus, it is reasonable to deduce that PSC-SN-promoted trans-migration of cancer cell by providing adhesive molecule(s) to the interface of inserts, which in turn facilitated cell adhesion and then accelerated the motility.
In vitro culture of human fibroblasts and keratinocytes showed that the zinc oxide nanoparticles facilitated cell adhesion, proliferation and infiltration of chitosan-collagen 3D porous scaffolds.
To investigate the viability of such scaffolds, HEF1 fibroblast cell assays were conducted and the results revealed that the nanotopography of the PCL-SK scaffolds facilitated cell adhesion and proliferation.
Among the three scaffolds, SA/COL could distinctly facilitated cell adhesion, accelerated cell proliferation and enhanced the expression of cartilage specific genes such as Acan, Col2al and Sox9 than the other two groups.
The CCL21/SLC facilitated cell adhesion in the presence of HMVEC-LLy, but only a small difference was found between the two cell lines in its absence (10.3 ± 9.7 versus 3.0 ± 2.1, p = 0.012).
Cell attachment assays with fibroblasts revealed facilitated cell adhesion on RGD modified block copolymer brushes.
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Signaling focal contacts via ITGβ3 were identified to facilitate cell adhesion, migration and mediate ECM-physical cues to modulate HSPC function.
Surface coating and pore formation were achieved to facilitate cell adhesion and cell growth.
Collagen type I was immobilized onto the scaffolds to facilitate cell adhesion.
This could be related to the SF amino acid sequence that acts as cell receptors facilitating cell adhesion and growth.
In order to culture the cells on the SPEs surfaces the working electrodes were coated with poly-l-lysine to facilitate cell adhesion.
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