However, Fos expression in the infralimbic cortex was positively correlated with reinstatement behaviour in the current study, indicating that this region is indeed activated and the extent of activation is related to the extent of the reinstatement of the conditioned approach to the CS.
We utilized a parallel plate flow system to investigate the effects of simulated arterial (500 s−1) and venous (50 shearshearateses and TM surface concentration (0 1400 fmol cm−2) on the rate and extent of activation of protein C. The rate of production of activated protein C increased with shear rate and TM surface content.
Although the reported results are after 16-h incubation, measurements just after the start of the incubation, and as a function of the length of incubation up to 16 h, showed that the FP-mediated increase in arginase activity was immediate and that the extent of activation was independent from the incubation time (data not shown).
Our results extend the idea that many genes with sex-differential expression are either activated or repressed as a consequence of DSXM and DSXF activity in both sexes, but that the extent of activation or repression is sex-specific [ 44].
Remarkably, in the spontaneous group very little increased recruitment induced by the presence of emotional words was present in the areas activated by the task, especially compared to the extent of activation of the dorsal attentional network associated with the explicit instruction.
In our previous microarray study examining modes of DSX-regulated gene expression at the adult stage in head tissues, a large number of sex-biased genes were found that were either activated or repressed as consequence of dsx activity in both males and females, but the extent of activation or repression was sex-specific [ 43, 44].
STAT3 is constitutively activated in 60% of primary high-grade/malignant gliomas and the extent of activation correlates positively with glioma grade.
The extent of activation of the AP-1 system is paralleled by a shift in activated cell types, from mainly endothelial cells in normal tissue to predominantly vascular SMCs in advanced atherosclerotic lesions.
The extent of activation was determined by analysing DNA adducts by 32P-postlabelling.
Depending on different genetic backgrounds and the extent of activation of these mechanisms, various pathologies with resulting symptoms can ensue.
The porous structure of ACs is a function of the precursor used in the preparation, the activation method followed, and the extent of activation.
