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The mechanisms causing the reduction of TRIM8 expression were explored by using qPCR and in vitro assays.
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The relationship between PDCD4 and miR-184 expression was explored by Pearson test.
The relationship between miR-148b and NRP1 (CST) expression was explored by Spearman correlation.
Differential miRNA expression was explored by unsupervised cluster analysis and was then associated with the molecular subtypes and genetic aberrations commonly present in breast cancer.
The impact of differential BCL2 expression was explored by comparing the HR for women with tumours showing varying levels of BCL2 staining intensity using a simple univariate analysis.
The protein expressions were explored by Western blot to clarify the mechanism by which AgNPs exert a cytotoxic effect on PC-3 cells.
Additionally, the putative mechanisms of Cygb expression in cancer were explored by subjecting HNSCC cell lines to hypoxic culture conditions and 5-aza-2-deoxycitidine treatment.
The importance of gains and losses for the expression of these genes were explored by including genomic microarray data in the analyses.
Global gene expression changes in our dataset were explored by principal components analysis (PCA) to reveal the relationships between the different cortisol delivery regimes [33].
The possible mechanisms of Gent activities were explored by evaluating expression levels of tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), IL-6 using real-time fluorogenic PCR and expression levels of cyclooxygenase-2 (COX-2), inducible nitric oxide synthase (iNOS) using Western blotting.
Then the possible mechanisms by which Zn prevents the aorta from diabetes-induced pathogenic changes were explored by analyzing Nrf2's expression and function, and also MT expression.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com