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Figure 6C indicates that cell populations with enforced miR-26a expression were characterized by significantly increased numbers of cells arrested in G1, which is more than that of tumor cells treated with miR-NC containing PLGA nanocomplex or untreated control (Table 3).
The LPS-induced cytokine production and cell surface marker expression were characterized by flow cytometric analysis.
Morphological changes caused by EGFP-MVaf1 expression were characterized by fluorescence microscopy, in Z-stack acquisition mode.
Levels of apoptotic markers, signaling molecules, and cell cycle regulators expression were characterized by Western blot analysis.
Genes with lineage-specific patterns of differential expression were characterized by a high frequency of SNPs associated with TSSs.
Nonetheless, a few genes with known gynoecium-enriched expression were characterized by MPSS that were not identified in previous microarray analyses, including three Embryo Defective and Embryo Defective-like genes.
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In confocal images of lumbar tissue stained for NeuN and NK1-R immunoreactivity (figure 3B G), NK1-R expression is characterized by appearance of an outline of NK1-R immunoreactivity surrounding NeuN immunoreactivity.
To study the two candidate genes for the development of NPC, mRNA expression was characterized by quantitative real-time PCR in 3 NPC cell lines, CNE2, SUNE1, and C666-1, in the immortalized normal nasopharyngeal epithelial cell line NP69, and in 11 primary NPC tissue samples with adjacent normal tissue.
Enzyme expression was characterized by electrophoresis and immunoblotting as a function of time in culture and phenotypic differentiation.
However, the most anterior domain of AmphiSyn expression was characterized by a cluster of ventro-lateral nerve cells.
Adenocarcinomas derived from the lung in addition to high caspase-8 expression are characterized by increased expression of DR4 compared with adjacent non-neoplastic tissues.
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