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T cell antigen receptor control of Myc expression was explained by TCR control of the frequency of cells that express Myc mRNA.
Manual milk expression was explained by a lactation consultant demonstrating the Marmet technique [ 9].
As a result, we were able to determine that up to 16% of the variance in gene expression was explained by considering the interaction between the variants, with an average additional variance explained of 4.3% (Table 2; Supplementary file 1B; Figure 3).
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This lower expression is explained by lower mRNA levels (Figure 6C).
Most (>80%) of the observed significant correlations of cis-regulated gene expression are explained by correlation of the underlying genotypes.
High statistical significance of the nitrogen fixation phenotype expression is explained by relatively low variability in up-regulation of 45 genes representing this phenotype (Table S2).
A relatively small proportion of over-all variation in gene expression is explained by differences between chimeric and clonal development.
Nevertheless, their inclusion in the model allows us to see to what extent their expression is explained by the proposed mechanism.
We find that a relatively small proportion of transcriptional variation in gene expression is explained by differences between chimeric and clonal development.
Within altered chromosomal regions, relations between gene expression and CNA level were assessed by Pearson correlation test (p < 0.01) on genes for which variations (at both level, gene CNA and deregulated expression), were explained by more than 10% of tumors.
We find, however, a relatively small proportion of over-all variation in gene expression is explained by differences between chimeric and clonal development, especially when compared to the large-scale changes in gene expression associated with the developmental process in this species.
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