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While integrin receptor α5β1 was overexpressed in tumor cells, its expression was corrected to the level found in control BEP2D cells after Betaig-h3 transfection.
The hGR MFI and hGRα mRNA fold change were significantly related to each other (r s = 0.64, p < 0.001), even when the hGR protein expression was corrected by the percentages of WBC monocytes (r s = 0.59, p < 0.01).
For each mRNA, gene expression was corrected by the rRNA11S content in each sample.
In all cases, expression was corrected with that of β-actin measured on the same sample in parallel on the same plate.
Gene expression was corrected against GAPDH and GUS mRNA level in each sample.
Protein expression was corrected for the expression of an internal control (Tubulin).
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In order to correct for hypothesis testing on such a scale, p-values relating to differential expression were corrected for multiple testing using FDR correction (Storey & Tibshirani, 2003).
Levels of expression were corrected for minor differences in loading.
The P values for differential expression were corrected for multiple testing using the method of Benjamini and Hochberg (1995).
P-values for differential gene expression were corrected for multiple testing using the Benjamini and Hochberg method [ 57], and an adjusted p-value threshold of 0.05 was used.
Also, birthdays of apoptotic cells and GPT neurons in ablated mice fit these patterns when lacZ expression is corrected to the earlier, more accurate, timing of HSV-TK expression.
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