Sentence examples for expression ref from inspiring English sources

In this study, we have investigated the tissue-directed expression of lysozyme in Drosophila using the UAS-gal4 system (41) and the two drivers Act5C-gal4 (ubiquitous expression; ref. 42) and gmr-gal4 (retinal expression; ref. 43).

The HMG boxes enable HMGB1 to bind chromosomal DNA and fulfil its nuclear functions, including determination of nucleosomal structure and stability, and regulation of gene expression (Ref. 49).

In gastric tumour specimens, miR-10b levels were dramatically elevated in lymphoma node metastasis-positive tumour tissues compared to lymphoma node metastasis-free tumour tissues and were found to down-regulate HOXD10 expression (Ref. 99).

Previously, we showed elevated BRCA1-IRIS expression in approximately 80% of breast tumors (>800 tumor samples were analyzed) that was correlated with elevated p-AKT and survivin expression (Ref. [ 38]).

Although it is not known how IRPs regulate CDC14A expression, this finding reveals interesting links between iron metabolism and cell cycle regulation, particularly in light of recent findings connecting iron-depletion-mediated growth suppression at the G1 S transition with a mechanism regulating cyclin D1 expression (Ref. 58).

Reactions were performed with primers specific for hsa-miR-146a (ref #001097) or hsa-miR-146b-5p hsa-miR-146b-5p hsa-miR-146b-5p hsa-miR-146b-5pwell plates at 95°C foref00468, followed by 40 cycles of 95°C for 15 s and 60°C for 1 min. theget gene expression wABInormalized based on the values of RNU44 RNA exPRISM®on (ref #001094).

The expression of miR-181c, miR-212 and miR-512 was silenced with DNA hypermethylation in gastric cancer, and their restored expression could induce decreased gastric cancer cell growth via inhibition of oncogenes expression (Refs 81, 82, 83).

Hypermethylation silencing miR-129 expression is associated with a poor clinical outcome in gastric cancer while restoration of miR-129 is linked to the cell growth inhibition and stimulation of apoptosis through suppression of CDK6 expression (Refs 79, 80).

MLL is required for proper segment identity in mammals, it displays haplo-insufficiency and regulates self-renewal of haematopoietic stem cells by controlling HOX (homeobox) gene expression (Refs 22, 23, 24).

The miR-200 family promotes EMT and can lead to cancer cell migration by impairing E-cadherin and ZEB2 expressions (Refs 73, 74).

On the other hand, ASC were shown to exhibit a more adipogenic gene expression pattern (Ref. 64), illustrated by higher expression levels of adiponectin and visfatin (Ref. 59).