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The expression of blood group antigens on the glycoproteins of differentiating Caco-2 has also been explored13.
Confirmation of CPT1AKD using a second shRNA, additional effects of CPT1AKD, silencing another FAO-related gene in LECs, CPT1A overexpression does not induce the expression of LEC markers and CPT1AKD in VECs does not change the expression of blood endothelial cell markers.
St Clair, EW, Wilkinson, WE, Lang, T, Sanders, L, Misukonis, MA, Gilkeson, GS, Pisetsky, DS, Granger, DI, and Weinberg, JB. "Increased expression of blood mononuclear cell nitric oxide synthase type 2 in rheumatoid arthritis patients". The Journal of Experimental Medicine 184, no. 3 (September 1996): 1173-1178.
Differential expression of blood group antigens on epithelial cells may influence tumorigenesis through altered glycosyltransferase specificity [7].
To examine how changes in the expression profiles of early mesodermal markers affect the differentiation of other lineages of mesodermal origin, we analyzed the timing and quantified the levels of expression of blood, endothelial and smooth muscle cell-specific markers.
Hence characterizing gene expression of blood cells seems to be the choice in this field.
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These alterations take place through several mechanisms that include aberrant glycosylation of mucin side chains, immunoreactivity of mucin core peptide, deletion of normally expressed antigens, expression of blood-group incompatible antigens, and de novo appearance of new antigens [ 2].
Differential gene expression of blood-derived cell lines in familial combined hyperlipidemia.
A 1.5 T MRI with gadolinium, focused on posterior fossa and temporal bone, was normal, except for a nodular area of contrast enhancement at left geniculate ganglion level, possible expression of blood-brain barrier damage or demyelination.
We performed a detailed temporal expression profile analysis of the putative trypsin transcripts using qPCR and confirmed the expression of blood-induced and blood-repressed trypsins.
Specifically Wnt7a but not VEGF promotes migration and stimulates expression of blood-brain barrier specific transporters of glucose (GLUT-1) in mouse brain endothelial cell line [ 82].
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