In recent years, many studies used microRNA expression profiles to classify human cancers and observed that expression levels of many microRNAs were under-expressed in cancers [ 18].
Comparison of gene expression revealed that retinoic acid receptor γ (Rar g) was predominantly expressed in NGN3+ cells, but not in GFRα1+ cells, whereas the expression levels of many other RA response-related genes were similar in the two populations.
In addition, the expression levels of many clones were improved in comparison to the parental enzyme.
Under drought, salinity and cold stresses, the expression levels of many genes involved in JA biosynthesis were up-regulated and those genes involved in JA signaling were differentially modulated.
Overexpression of any of these genes alone did not result in a high yield comparable to fadR expression, indicating that FadR enhanced fatty acid production globally by tuning the expression levels of many genes to optimal levels.
Surprisingly, recent studies combining the analysis of gene expression (transcriptome) and DNA methylation (methylome) using whole-genome tiling arrays revealed that DNA methylation regulates the expression levels of many ncRNAs.
It is more likely the case that assumptions about consistent expression levels of many housekeeping genes are incorrect.
In that regard, it is interesting to note that in our experiments we also detected changes in the expression levels of many of these genes whose protein products are regulated by phosphorylation events.
The expression levels of many genes in this latter group reflect the leakiness of the mutant lines, since a proportion of each mutant parasite population (20 30%, Figure. 3A) are capable of differentiating into bradyzoites.
We found that in uterine ULMs the levels of the most dysregulated microRNAs show an inverse association with the expression levels of many predicted target genes, and that they may affect multiple homeostatic pathways and functions.
