Although there are dissenters — e.g., Ziff (1974) — the claim that natural languages contain infinitely many complex expressions is plausible.[17] But it is equally plausible that nobody who reads this entry the first time has ever encountered this very sentence before, and consequently, the detour through cardinality considerations seems superfluous.
Given the level of typical inter-culture variation in gene expression, it is plausible that there is no specific response to Tf as an iron donor, which is consistent with its wide distribution within the host.
Given the reduction in expression, it is plausible that the 1459 EVs carry miRNA acting to down regulate key proteins in RAF-MEK-ERK mediated signaling.
Given the variety of NEP target peptides and their wide expression pattern, it is plausible that MME deleterious variants may contribute to the aetiology of different neurological conditions, and further studies are needed to identify the mechanisms underlying the phenotypic differences.
Although the two non-neuronal tissues we have tested here have provided limited utility in identifying SZ-associated gene expression abnormalities, it is plausible that very large case-control samples (to reduce individual variation/SZ heterogeneity) of LCLs or fibroblasts may be suitable for validation (e.g. by qRT-PCR) of a small number of genes identified via other means.
Of note, among our tumours with active Src, 85.3% had PTEN-preserved expression; therefore, it is plausible that in some cases PTEN was non-functional.
However, influences such as essentiality [ 32] or transcription interruption [ 33] are involved for E. Coli as well as expression levels so that less than 100% of gene expression "with the flow" is plausible when considering the relationship between replication and transcription for mouse.
Our data provide genetic and biochemical support for the concept that ABCA2 expression is a plausible contributory factor to the multifactorial mitoxantrone-resistant phenotype, while for other transporters no apparent changes in the expression could be correlated with mitoxantrone accumulation or cytotoxicity.
Although the mechanism for these observations remain unclear, the notion that fluoropyrimidine sensitivity might be modulated through either TS expression or p53 mutation is plausible [ 13].
Although studies on the role of AGR2 in promoting MUC expression are lacking, it is plausible that AGR2 could promote breast cancer metastasis through the regulation of MUCs.
