Given that TRX is involved in DNA and protein repair (Lu and Holmgren 2014), it can be deduced that the down-regulated TRX expression in this study indicated less DNA and protein damage.
The GUS expression in this study revealed both by histochemical and fluorescence assays in pSH4-GUS transgenic plants indicated the multiple tissue-specific expression pattern regulated by pSH4, as expected from the characterization of this promoter.
A C-terminal c-myc tag was incorporated (underline); this tag was incidental for H. capsulatum expression in this study, but was used in P. pastoris expression as described below.
Because TLR3 contributes directly to the inflammatory response of respiratory epithelial cells to both influenza A virus and dsRNA, we focused on the function of dsRNA in the regulation of COX-2 and IL-32 expression in this study.
Remarkably, the major variable affecting expression in this study was predicted reduced stability in the region surrounding the initiation codon (−4 to +37; where the A of the AUG codons is +1).
It is important to consider that the measurement of gene expression in this study represents the situation at a single moment in time, i.e. at the end of the training experiment when significant growth enhancement had occurred.
However, it did not show differential expression in this study.
We cannot exclude clinical treatment benefit for any level of HER2 expression in this study.
There are some limitations concerning about the evaluation of HER2 expression in this study.
The frequency of COX-2 expression in this study was 55.2% (Table 1).
