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GR regulates a large variety of gene expression in responses to glucocorticoid hormones [3], [4].
A role for Gnb1 expression in responses to multiple drugs of abuse is supported by the expression data.
When CskAS OTI CD8+ T cells were stimulated by APCs preloaded with peptides of different agonist potency during strong CskAS inhibition (5 μM), we observed marked augmentation of CD69 expression in responses to weak (T4, Q4H7, G4) agonists, contrasting with only slight enhancement of the responses to strong (OVA, Q4R7) agonists.
A variety of different platforms and approaches have been developed that have allowed analysis of gene expression in responses to viral infections [ 1- 6] Prior to the sequencing of the chicken genome these microarrays consisted of cDNAs from many different sources.
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Gene expression in response to Strep.
SA-mediated transcriptional responses control the temporal patterns of gene expression in response to stress.
iNOS expression in response to sepsis has been detected in neuronal and glial cells [71, 72].
We next examined the mechanism regulating NGF expression in response to cold challenge.
Functional changes in collagen type I protein expression in response to MSC were confirmed by ELISA.
By contrast, COX-2 expression in response to exogenous stimuli is robust in quiescent cells.
By histology and immunohistochemistry, we demonstrated that changes in gene expression in response to Strep.
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