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Downregulation of apolipoprotein (apo) C-III gene expression and upregulation of lipoprotein lipase (LPL) gene expression have been suggested to explain the hypolipidemic action of fibrates.
Differences in patterns of gene expression have been suggested as one of the contributing factor in sexual dimorphism in neuropsychiatric disorders [6].
Among such possibilities, pharmacological interactions between radiotracers and L-DOPA and/or L-DOPA-induced reduction of DAT expression have been suggested [10], [30].
Many genes in these regions encode proteins involved in the transcription and translation machineries, and gene dosage effects on expression have been suggested to limit their chromosomal location close to the origin of replication [18].
Changes in gene expression have been suggested to underlie many differences in gene function or in phenotype.
Alterations in miRNA expression have been suggested to play important roles in tumorigenesis and cancer progression [ 10].
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Coincidence between TRPA1 and neuropeptide expression has been suggested [72, 73], although evidence for coexistence of isolectin B4-positive and non-peptidergic neurons with TRPA1 has also been reported [74, 75].
By normalized methods, a wide range of non-dimensional parameters were considered and limit bending moment expression has been suggested which shows that r/t is the main factor affecting the limit bending moment.
Deregulation of its expression has been suggested to play a role in the development of several types of cancer.
Co-regulation of expression has been suggested to be among the causal factors for the clustering of biosynthetic genes in fungi [29], [30].
Between the two isoforms GAD67 is responsible for over 90% of basal GABA synthesis in the brain and its expression has been suggested to be regulated by activity [33], [34].
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