However, gene expression analysis displays that early innate immunity signatures including pro-inflammatory cytokines (TNF, IL-1β, IL-6), chemokines (CCL2, CCL3, CCL4, CXCL1) and neutrophil infiltration are strongly associated with acute death of the mice infected with IAV (Brandes et al., 2013).
Our present work showed that androgen-independent DU145 and androgen-dependent PC-3 and LNCaP PC cell lines tested using expression analysis displayed aberrant expression of LEDGF and Hsp27.
The Kaplan Meier analysis displayed that high expression of RRM2 had a negative impact on the OS (overall survival) and PFS (progress-free survival) of CRC in both sets significantly.
Microarray analysis and real-time PCR data analysis displayed that the deletion of Cdc42 decreased dHand expression level.
The microarray analysis displayed that control and hypertrophied EHTs differed distinctly in their expression patterns.
RT-PCR and Western-blot analysis displayed that compounds L1, L3, L5 (N-allyl-2,3,4-triallyloxylycoricidine) exhibited antiviral activity, which was evidenced by reducing TMV-CP gene replication and TMV-CP protein expression.
No brain phenotypes are known, but our expression analysis shows that CGNT2 displays a distinct spatiotemporal expression pattern suggestive for a function during brain development.
Here, we show by comparative expression analysis that, unlike for erythroid, granulocyte/macrophage or lymphoid lineages, mast cells display comparable expression levels of a whole set of blood stem cell-affiliated TFs.
Taken together, the gene expression analysis demonstrated that G-2 cells and tumors display more similarities than differences in their gene expression program; the differences are mainly related to their different biological context.
Quantitative ALK mRNA expression analysis confirmed that PAX3/7-FOXO1-positive ARMS tumours (ARMS-t) displayed higher ALK mRNA levels than fusion gene-negative RMS, with median expression values of 1405 (min.=10/max.=13 370 units) vs 23 (min.=1/max.=1614 units), respectively.
However, it is clear from this more detailed expression analysis that VvSTS6 and VvSTS16 display a similar pattern of induction in response to these three stress treatments and that the pattern of induction of these group A and C genes is clearly different to that observed for the group B gene, VvSTS48.
