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In Drosophila, cells that express corazonin also express receptor molecules for two unrelated diuretic hormones, DH44 and DH31 [9].
Mature osteoblasts express receptor activator of nuclear factor-κB ligand (RANKL), a protein that will signal through RANK, a member of the tumor necrosis factor receptor superfamily present at the surface of pre-osteoclasts, and induce differentiation of these cells [4] [7].
Clinically, 27% of primary tumors express receptor activator of NF-κB [ 146].
Cells that do not express receptor α can be sensitive to the complex of cytokine/soluble cytokine receptor α.
During the immune response, mature dendritic cells express receptor activator of nuclear factor-kappa-B (RANK) and tumor necrosis factor receptor-associated factor 6 (TRAF6) [ 15, 16].
Osteoblasts and stromal cells express receptor activators of the nuclear factor-kB (NF-kB) ligand (RANKL) and macrophage colony-stimulating factor (M-CSF) [ 2].
Similar(43)
MC express receptors for TGF-β1, and COX-2 expression can be induced in MC.
T cells express receptors for neuropeptides that mediate immunological activities.
Furthermore, it has been reported that human dermal fibroblasts express receptors for β-glucans like macrophages.
Objective: Most human endometrial and ovarian cancers express receptors for luteinizing hormone– releasing hormone.
Eighty percent of human ovarian and endometrial cancers express receptors for luteinizing hormone releasing hormone (LHRH-R).
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