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Exposure to polybrominated diphenyl ether (PBDE) flame retardants is widespread, with 97% of Americans having detectable levels.
To assess the impact of prenatal exposure to polybrominated diphenyl ethers (PBDEs) and polyfluoroalkyl chemicals (PFCs) on early infant neurobehavior.
Human serum and mother's milk are frequently used to assess exposure to polybrominated diphenyl ethers (PBDEs), including transplacental transfer to the foetus.
Lactational exposure to polybrominated diphenyl ethers and its relation to social and emotional development among toddlers.
Another study, also published in May, found that a mother's exposure to polybrominated diphenyl ethers, or PBDEs, during early pregnancy and after a baby is born could harm her child's developing brain.
Human exposure to polybrominated diphenyl ethers (PBDEs) is virtually universal in the United States.
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Human fetal exposures to polybrominated diphenyl ethers (PBDEs) and their metabolites (OH-PBDEs) are unique from adults, and in combination with a different metabolic profile, may make fetal development more sensitive to adverse health outcomes from these exposures.
background: California children's exposures to polybrominated diphenyl ether flame retardants (PBDEs) are among the highest worldwide.
Elevated exposures to polybrominated diphenyl ether (PBDE) flame retardants have been linked to developmental neurotoxicity in children and endocrine disruption in adults.
The aim of this study was to determine human prenatal and postnatal exposures to polybrominated diphenyl ethers (PBDEs), polychlorinated biphenyls (PCBs), hydroxylated metabolites of PCBs (polychlorobiphenylols; OH-PCBs), and pentachlorophenol (PCP).
Lorber, M., Exposure of Americans to polybrominated diphenyl ethers.
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