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Any exposure to CCP showed a significant relation with incident asthma, but no obvious increase in risk with increasing CCP exposure was detected.
Any exposure to CCP was also significantly related to the risk of incident asthma (OR = 1.66; 95% CI, 1.03 2.66).
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Our finding of increased risk of incident asthma in relation to CCP exposure in office workers is also original, but consistent with the observations from the Helsinki Office Environment Study, which showed significantly increased risk of wheezing (OR = 1.29; 95% CI, 1.06 1.56) and chronic cough (OR = 1.43; 95% CI, 1.14 1.78) related to handling of self-copying paper (Jaakkola and Jaakkola 1999).
Finally, a model including any exposure to paper dust, CCP, and/or FPP (≥ 1 hr/week) was fitted.
The question on exposure to paper dust, CCP, and FPP asked the study subjects to assess their average exposure in hours per week in their current job.
The aim of this study was to assess the relations of exposure to carbonless copy paper (CCP), paper dust, and fumes from photocopiers and printers to adult-onset asthma.
Including this four-CCP structure in interpretation of scattering data for the longer recombinant segments, CCPs 10 15 and 8 15, implied flexible attachment of CCPs 8 and 9 to CCP 10 but compact and intimate arrangements of CCP 14 with CCPs 12, 13 and 15.
Exposure to any of the three exposures at office work (CCP, paper dust, FPP) was related to a significantly increased risk of asthma (OR = 1.90; 95% CI, 1.05 3.44) (Table 2).
We recently reported an association between occupational exposure to mineral oil and RF-positive RA and anti-CCP-positive RA, respectively, among men [ 18].
Exposure to sucrose or sucrose-conditioned cues immediately after training enhanced retention of information required for expression of a CCP in the radial maze.
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