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Prolonged exposure of tissue to freezing temperatures causes tissue damage known as frostbite.
Such progress has been made possible by a number of factors including advances in transducer design, more accurate measurement and calibration of acoustic power and careful experiments to determine the precise nature of chemical processes taking place during and following the exposure of tissue to ultrasound.
Finally, it was shown that the cell survival/CEM43 relationship for the cases investigated here most closely aligns with isothermal exposure of tissue to temperatures of ~50 °C.
For instance, exposure of tissue factor (TF) and activation of TF-induced thrombin generation is now also considered to play a key role in thrombi formed in the arterial circulation [4], [5].
Our data extend earlier findings, using the same model of acute atherothrombosis, which indicated that thrombus formation is the result of a multi-factorial process involving: platelet interaction with collagen, exposure of tissue factor, generation of thrombin, and formation of fibrin fibers [26].
High- and lower-risk surgical procedures will be defined by exposure of tissue corresponding to the respective risk level during surgery.
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Hydrolysis, namely exposure of tissues to endogenous water resulting from inadequate tissue processing has a major impact on antigen degradation [8].
Notwithstanding, exposure of tissues containing either peripheral or central sensory nerve terminals to TRPA1 agonists invariably results in a calcium-dependent release of SP/NKA and CGRP.
For this reason, it is possible that possessing calcium carbonate skeletons might impart advantages in terms of uncoupling of the exposure of tissues to PAR and UVR.
Unremitting exposure of tissues to ROS can lead to pathological conditions, such as neurodegenerative disorders and chronic obstructive pulmonary disease [3], [4], [6].
Since unremitting exposure of tissues to H2O2 and other reactive oxygen species can lead to several degenerative disorders and diseases, our results have important implications for the use of NF-κB inhibitors in therapeutic drug design.
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CEO of Professional Science Editing for Scientists @ prosciediting.com