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The increase of lamp2A protein levels evidenced after ammonium chloride exposure may be interpreted as a compensatory reduced degradation of lamp2A in response to the presence of CMA substrate accumulation, as already reported [ 3].
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The mechanism for the protective effect of Kupffer cells was explained in part by a Kupffer cell dependent induction of hepatic transporters, particularly Mrp4, which may be interpreted as a defense against exposure to toxic compounds (Campion et al. 2008).
This may be interpreted as arising from risk-factor exposure at the group level (e.g. PFOA concentrations in the drinking water assumed to be constant within a particular water district).
In addition, the caption on page A255 may be interpreted as implying causality between prenatal exposure to PFOS and PFOA (perfluorooctanoic acid) and altered body weight and head circumference in human infants.
Adaptation may be interpreted as perceptual learning that results from exposure to discrepancy.
Additionally, all risk factors examined except TV watching, although assessed cross-sectionally by definition, may be interpreted as derived from a retrospective cohort, since the exposures undoubtedly have preceded the outcome.
This may be interpreted in line with models explaining adverse development in children as a result of exposure to multiple risk factors [ 51].
Then, the data may be interpreted.
These problems may be interpreted in terms of fuzzy numbers.
Of course, it may be interpreted differently by others.
This question may be interpreted in different ways.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com