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In contrast, the evidence base for postexposure prophylaxis (PEP) following sexual exposure is weak, with supporting preclinical and cohort data only (mainly occupational) [ 7].
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The relationship with noise exposure is weaker than for men.
We suggest that the difference of a few days is unlikely to be important, particularly since the effect of time (and therefore age) on the key electrical parameters that were changed by acid exposure was weak or absent.
Thirdly, the exposure was weak as many women stopped taking their prescribed trial medication.
Second, the major exposure factor (recreational drug use) was self-reported, the assessment of exposure was weak, and underreporting of the condition was possible in settings where using illegal drugs may be severely punished.
However, the response of the 10-day-old plants to the single ethanol exposure was weaker.
Associations with paternal occupational pesticide exposure were weaker and less consistent.
In line with other studies [ 7], the effect of traffic exposure was weaker for asthma compared to wheeze.
The association between same-day 24-hr average O3 and wheeze, controlling for particle exposure, was weaker but significant for the overall group of infants in the study.
Because the performance of the biomarkers in the classification of the total arsenic exposure was weaker than in the classification of iAs alone, some low-toxicity organic species may have diluted the biological responses to arsenic.
As a result, the pattern of expression observed was very similar between Q-RT PCR and RNAseQ-RT PCRs in 18 of the 20 genes andlysed, while foRNAseqic_t_481778 the induction afteresults of cold exposure was weaker by Q-RT–PCR than by RNAseq, and for OlePin_t_288184 the pattern was ofpositheat 10 days of cold exposure (Fig. 10).
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CEO of Professional Science Editing for Scientists @ prosciediting.com