Sentence examples for exposure figures from inspiring English sources

There was no evidence of apoptotic induction in the HT29 cell line after 4 and 8 hours of exposure (Figures 3F and 3H respectively).

There was no evidence of cell cycle progression in PHA-stimulated PBLs exposed to LD, however viable cells were still present after 72 hours of exposure (Figures 9D, E and 9F). Figure 9 A-F: Histograms showing PI stained non-stimulated and PHA-stimulated PBLs exposed to LD at 1 10 for 24, 48 and 72 hours.

For non-stimulated primary blood lymphocytes (PBLs) exposed to 1 10 LD (made from 3 ml/30 ml stock) there was a decrease in the number of viable cells in G0 as time passed, however viable cells were still present after 72 hours of exposure (Figures 9A, B and 9C)).

Exposure resulted in a significant reduction in the number of AV after 3 hr of exposure (Figures 2B and 2C, 53.5±2.8), which remained low following 24 hr of exposure (Figures 2B and 2C, 48.6±2.5).

Another intriguing finding of the present study is the observation that insulin stimulated IRS-1 serine phosphorylation in the absence of T, while concomitantly inhibiting the phosphorylation AKT beyond 30 minutes of insulin exposure (Figures 2A and 3A).

Consistent with the notion that etoposide induces an aberrant DNA structure in the BCR, resulting in a different chromatin configuration as marked by increased RPA and RAD51 levels, the level of INO80 also increased in the BCR compared to the control locus upon etoposide exposure (Figures 5A and B).

The fraction of cells switching to the planktonic state was significant after 2 h of exposure (Figure 1c), but not significant after 1 h of exposure (Figure 1d).

The exposure figure is related to the way that the survey is setup.

Additionally, the hatching rate of the zebrafish embryos was influenced by TiO2-NPs exposure (Figure 5).

The histological sections also showed that Bcl-2 accumulated in the nuclei after radiation exposure (Figure 4).

Laser scanning confocal microscopy (LSCM) was used to measure the melanocytes in forearm skin after ultraviolet exposure (Figure 1).