Sentence examples for exposed to inflammation from inspiring English sources
However, in SpA, the bone tissue directly exposed to inflammation (osteitis) is the trabecular bone of the vertebrae, but not the cortical bone surface as in RA (synovitis).
Together with our observation that soluble GRP78 can act as a ligand and activator of sGRP78, these results indicate that beta cells trigger a self-destructive feedback loop when exposed to inflammation, actively participating in their own demise.
However, the fact that MSCs have been directly exposed to inflammation in the joint has not been given sufficient attention.
Cells undergoing mitosis, identified by pH3 immunoreactivity, were not significantly different in number between controls and animals exposed to inflammation (Fig. 6D).
This study demonstrates that aberrant expression of the Rxra-centered transcriptional pathway in Leishmania-infected KCs favors parasite survival and validates the use of cells exposed to inflammation as a means to discover pathogen-regulated transcriptomic pathways in vivo.
We hypothesized that it might be possible to use the transcriptomic response of uninfected cells exposed to inflammation as a measure of the baseline or normal in vivo response to insult.
Reversing behavioural abnormalities in mice exposed to maternal inflammation.
Altogether these findings advocate for a role of these immune mediators in the morphological and neurobehavioural changes in offspring exposed to prenatal inflammation.
Therefore our results could suggest that this LPS-induced fetal synaptic damage may contribute to the altered neurobehavioural state often described in children exposed to antenatal inflammation by modulating neuronal connectivity.
Patients with RA are not only exposed to systemic inflammation from their arthritic disease, the failing heart itself may be invaded by peripheral blood mononuclear cells that participate in left ventricular remodelling.
As described earlier, the native cartilage is exposed to chronic inflammation conditions by increased levels of inflammatory mediators including IL-1 β, TNF- α, and prostaglandin E2 (PGE2) [ 87, 88].
