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Genotyping of Mycobacterium tuberculosis isolates is a powerful tool for epidemiological control of tuberculosis (TB) and phylogenetic exploration of the pathogen.
Among others, detection of such variability can play a major role in driving exploration of the pathogen's biology and in understanding the development of resistance to drugs.
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Human challenge studies could allow further exploration of the host and pathogen factors associated with the iNTS phenotype; however, the unpredictable response to a controlled challenge in healthy volunteers is a potential hurdle.
Thus, plenty of scope remains for further exploration of vector specificity by subpopulations of the pathogen.
Studies of coinfections with pathogens that impact humans have provided the most comprehensive exploration of the change of effects of coinfecting pathogens.
The F. graminearum GeneChip will help to accelerate exploration of the pathogen host pathways that may involve interactions between pathogenicity genes in the fungus and disease response in the plant.
As a contribution to this baseline, we outline approaches, protocols, and empirical models derived from a decade of exploration of pathogen biodiversity and disease in northern ecosystems (Table 1), illustrated with examples of drivers for emerging parasitic disease in northern wildlife (Table 2).
Shipments of the pathogen continued through April 2015, Reuters reported.
The joining of the pathogen to the antibody sets off a cascade of actions.
Cows and their manure are considered the major sources of the pathogen.
There is a single mention of the pathogen before October 2012 on the Defra website.
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