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A possible explanation for these differences might be the different cell systems used.
A possible explanation for these differences could be the different type of patients in each study.
One intriguing explanation for these differences might be that telomerase uses different mechanisms to interact with different sequences of telomeric DNA.
The most likely explanation for these differences is that we used different mapping populations.
The explanation for these differences may lie in the different types and/or activation status of cells used for the experiments.
One explanation for these differences may be technical reasons, for example, different methodologies (including antibody source) and different fixation and processing methods between experimental and human tissues.
The possible explanation for these differences can be due to the different study populations and settings.
A possible explanation for these differences could be found in the different assessment protocols used.
One possible explanation for these differences can be attributed to the different methods used for the assessment of TIMP3 expression.
The explanation for these differences may be related to the different forms the health markets have taken in the two localities.
The main explanation for these differences may be a dependence on the different behaviour of the discretized integrals and noise contaminated with real data.
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CEO of Professional Science Editing for Scientists @ prosciediting.com