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There are several possible reasons explaining this difference, including the use of different hardware, absence of standardization of image acquisition (choice of different b values), various methods for ADC calculation and different patient populations.
Our study does not allow explaining this difference, but we suppose that patients with anxiety or depression may have different psychological mechanism than patients with somatoform disorder to cope with these stressors.
The lower number of air travellers coming into Italy per year compared to US and UK (25 millions, versus 73 and 92 millions, respectively) [9], could also play a role in explaining this difference.
One hypothesis explaining this difference between the two culture methods is that, in the chemostat, DO was limited.
A potential mechanism explaining this difference is that the timing of food intake can influence the circadian system [ 44].
To explore the likely role of chance in explaining this difference, an unpaired t-test can be performed.
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The different patient characteristics and different trial characteristics within these trials may explain this difference.
They also suggest that different exposure routes and metabolic rates could explain this difference.
However, different generators and circuits involved in CNV and LEPs could explain this difference.
Franken (even in academic robes) might explain this difference.
What explains this difference?
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