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Cua et al. explained this paradox by the double usage of the IL-12 p40 subunit by both IL-12 and IL-23 (heterodimer of IL-12p40 IL-23p193p19 subunits).
We have partially explained this paradox by demonstrating that increased bio-availability of IL-2 upon daclizumab treatment predicted by mechanistic in-vitro studies 28 activates CD56bright NK cells 19, 28 and common ILC precursors 18 expressing high levels of intermediate affinity IL-2R unhindered by daclizumab.
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Both artificially inflate death rates among those of normal weight and help explain this paradox.
To explain this paradox, this Article analyzes the legislative process surrounding recent European legislation aimed at facilitating cross-border voting.
To explain this paradox, this Article first tries to assimilate the new education reforms to the most frequently proposed alternatives to bureaucratic governance — marketization, managerialism, professionalism and craft.
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