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The higher than average family size we observed in multiple extant pathogens that form a monophyletic clade in the species tree, are explained by gains in the common ancestor of these related species, maintained in the extant species (DOLLOP and CAFE).
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The higher risk of mortality associated with gain in WC may e.g. be explained by gain in harmful abdominal fat or by loss of beneficial peripheral fat or lean body mass that may accompany the WC gain as changes in BMI are fixed.
We also investigated the effects of being first-, middle-, or lastborn on a son's probability of becoming a landowner, to test the possibility that any higher success among firstborn sons may be explained by gaining access to higher resources at reproductive ages.
This fact can be explained by increase the flat gain length in the interval of fundamental frequency that makes methods more sensitive to noise.
The other four introns can be parsimoniously explained by single gains with no losses.
Their non-universal distribution in the group of Lactobacillaceae species considered can be explained by differential gains and/or losses, which at present cannot be resolved.
In principle, if there were few protosplice sites in each gene, the presence of introns in the same positions of orthologous genes in distantly related species could be completely or at least to a large extent explained by parallel gains.
The increase in insulin resistance could not explained by weight gain or alterations in lipid levels.
This may be explained by gene gain at an early phase of monocot radiation, or alternatively by gene loss in the ancestor of the eudicot lineage.
Our results suggest that auditory selective attention in humans cannot be explained by a gain model, where only the neural activity level is increased, but rather that selective attention additionally enhances auditory cortex frequency selectivity.
The current experiment shows that auditory selective attention in humans cannot be explained by a gain model, where only the neural activity level is increased, but rather that selective attention additionally enhances auditory cortex frequency selectivity.
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