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This study also helps explain why the aggregates formed when autophagy is compromised are decorated by antibodies to both ubiquitin and p62.
This may be an additional factor that explains why the aggregate number changes more dramatically than the SDS-soluble mutant huntingtin.
This would explain why the distribution of GBR-IR signal in this region appears in coherent aggregates larger than the C2 terminals and larger than the Cha-Gal4-expressing boutons (Fig. 6E).
These 'thick' boundary conditions explain why the solution converges fast.
We will explain why the conditions are nearly optimal and give an application.
To explain why the fluence required for efficient perforation is only 20 times higher instead of 100 as predicted by theoretical absorption curves, it is tempting to assume the NPs aggregate.
Considering this observation together with the activation of the UPR may help to explain why the HTorAΔE-expressing flies had similar life spans to those of the HTorAWT-expressing flies and why they did not manifest severe dystonic symptoms under normal conditions despite the presence of protein aggregates at the NE, the ER and the mitochondria and structural defects at the synapses [ 29, 30].
The low levels of ATP explain why the synoviocytes die.
Does it explain why the code is triplet?
This would explain why the alignment cannot be scrolled softly.
This may explain why the correlation studies failed.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com