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At least two potential mechanisms may explain this modification of the self-reactive repertoire observed.
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Also, food preparation methods may partially explain this effect modification.
We were unable to explain this effect modification, although unmeasured factors such as cultural or biological characteristics may have been responsible.
In order to explain this behavior, a modification of the energy balance, which takes into account the presence of plastic deformation when martensite coexistence occurs, has been proposed.
A higher incidence of cirrhotic patients in group A2 and more frequent ribavirin dose modification could explain this behavior.
The association of H3-K56 acetylation with the nucleosome deposition machinery mechanistically explains how this modification associates with progressing replication forks and with promoter nucleosomes that display high turnover [6], [18].
Should the changed editorial have carried an editor's note to explain the modification?
We describe the fractional step methods first assuming the former, and then we explain the modifications introduced by the stabilized formulation we employ and that has been proposed in a previous work.
In addition, the absence of any significant correlation between variations in ToM performances and variations in executive scores indicates that possible impairment of the patients' executive functioning cannot explain the modifications in ToM performances in the postoperative condition.
All of these mechanisms have previously been suggested to explain the modifications induced by experimental pain[ 11, 14, 18].
Increased fatty acid synthesis or reduced fatty acid oxidation resulting from fetal hyperinsulinemia and high Δ6-desaturase activity might explain these modifications, as we have previously suggested as discussed elsewhere [ 32, 35].
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