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In order to explain this incompatibility, several researchers have claimed that classification of tumours on the basis of HER-2/ neu overexpression or amplification may define a subset of breast cancer in which the net effect of a risk factor could be rather more obvious and its impact on breast cancer development more clear [ 3, 7, 8].
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Not to mention being obliged to explain the incompatibility of quantum mechanics and gravitational theory to dinner guests on Hawking's behalf, using peas and potatoes.
Our results are consistent with these dominance effects; however, if faster-male effects solely explain the incompatibilities in this cross, then hybrid females are not expected to suffer from incompatibilities either.
How to explain this?
Two things explain this.
Can you explain this?
What might explain this?
Interestingly, the results of the microsatellite analysis suggested the absence of transmission ratio distortion between paternal and maternal alleles even in the offspring of the AUS crosses with either ARG and/or MOZ strains, reducing the possibility of parental genetic effects to explain strain incompatibility.
What explains this disconnect?
What explained this?
What explains this inertia?
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CEO of Professional Science Editing for Scientists @ prosciediting.com