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Immigration and short time elapsed since outbreaks could explain the conservation of genetic diversity after the demographic crash.
According to our model of cis-regulated alternative splicing, splicing variants would be kept in the population due to balancing selection acting on tissue-specific variants, which would explain the conservation of the splicing status between human and mice.
On one hand, they suggest that coexpression per se cannot explain the conservation of bipromoter structures.
The imprinting of Pdcd2 and neighboring loci could explain the conservation of the synteny.
However, it is not sufficient to explain the conservation of spatial proximity over a longer period of time.
We can reasonably assume that regional genomic propensities detected by the mutual information measure at least partly explain the conservation of phylogeny sizes between populations.
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The sequence of this loop appears extremely conserved within Staphylococcaceae SRP sRNAs, and also well conserved in the Firmicutes phylum, indicating a common evolution which explains the conservation of the loop structure.
This finding explains the conservation of the axoneme-specific sequence motif through 1.5 billion years of evolution.
Although speculative, this perhaps explains the conservation of Asp-55 and Asp-56 along with Asp-57 (Fig. 6b).
This second phase of evolution explains the conservation of the new surfaces among Mecopterida species.
In contrast, the preservation of structure at the 3′-end of the helix explains the conservation of nucleotide preferences at adjacent sites of the binding motif.
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