Variation of phenotype strength may explain frequencies of mutant alterations sharply below 25% within some crosses (curly, levi, smerz) (Table 2).
A logical hypothesis to explain frequency-specific adaptation is that adaptation is occurring at or before the inputs to the recorded neuron.
The high frequencies of ipaBCD and ipaH genes could explain frequencies of fever, vomiting, and dehydration in infected children.
To further examine this, a Log Linear Model was constructed which attempted to explain frequencies of quinolone TRAs based on expected antibiotic exposure and host association.
NI struggles with the concept of frequency or does not explain that frequency changes across generations.
Although associations do reflect the frequency of specific alleles in different countries, the differences among countries cannot completely explain disease frequency.
This parameter is however shown to be insufficient to explain higher frequency effects.
Under some circumstances, four is the optimum number, which could explain the frequency of tetrameric enzymes in nature.
In particular, the diffusive time scale is used to explain the frequency dependence observed in electroporation experiments with uni- and bi-polar pulse trains.
