Susceptibility to schizophrenia is explained for the largest fraction by genetic variation [1].
Heterosis, increased hybrid vigour in early-generation hybrids (Rieseberg et al. 2000; Johansen-Morris and Latta 2006), probably explains, for the larger part, that all BC1S1 families produced at least some seeds, even though these hybrids where backcrossed once to one of the parents.
However, also absence of selection pressure (neutral mutations) could explain for large variation within genes.
Explained variance (R) for the model was.51 (which means that the dependent variable is explained for a large part by the identified independent variables, but also by some, as of yet, unidentified variables).
All the QTL that showed an interaction with diet accounted for a larger proportion of variance explained for the HE diet than for the LE diet.
Soil structure and interactions between biotic and abiotic processes are increasingly recognized as important for explaining the large uncertainties in the outputs of macroscopic SOM decomposition models.
This study presents a microscopic picture at atomistic details for explaining the large difference in the dissociation rate constant among different inhibitors, which might provide important guidelines for design of the novel potent inhibitors with optimized interactions.
These relations provide a framework for explaining the large interspecific variation in physiological and life-history variables.
Analysis of all differentially present genes is of utmost importance, since these genes denote the accessory genome, which most likely represents the genomic repertoire that explains for a large part observed virulence, pathogenicity or transmission differences between clones.
This bias was not controlled for and may explain the large value of α measured, notably compared to the females.
