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In vitro experiments indicated that while NKD1 induction is a direct result of β-catenin signalling, FZD3 expression requires a Wnt growth factor signal.
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The experiments indicate that while it is difficult to construct models using only historical data that consistently perform well, there are models that show good performance under certain pre-defined conditions, and that many of these models have an interesting property.
Taken together, these experiments indicate that while Vav proteins are essential for the induction of T cell proliferative responses, the intrinsic GEF activity appears dispensable for Vav function in T cells.
Control experiments indicated that the plasticity, while requiring sAPs to be induced, was expressed independently of synaptic activity since changes persisted when sAPs were blocked.
Indeed, while competitive experiments indicated that EBP prevails in mediating SR31747A antiproliferative activity, an analysis of the expression of both receptors indicated that the cellular sensitivity to SR31747A is not correlated with either EBP or SR-BP expression.
While our experiments indicated that EPO-induced cardioprotection from ischemic damage is not mediated by NO activation, it had been recently reported that EPO-induced NO release requires the integration of EPO receptor and cβR with activation of Akt and PI3 kinase signaling (Su et al., 2011).
In two cases we observed selection on the expression of plasticity by E. brachycarpum in the field while controlled environment experiments indicated that plasticity was selectively neutral.
Field experiments indicated that the maximum cutting depth was 200 mm while the average width of slits was 12.8 mm.
The results of our experiments indicated that the hybrid ensemble shows the worst performance while the MLE ensemble provides superior performance in most cases.
Preliminary experiments indicated that Cu(IDA -capped lIDA -cappedignificant celipidsicity, whade cell culture conditionsignificantelt MNPVs cellosed of 10:1 DPPC/DMPC.
The turbidity experiments indicated that the WT protein began to aggregate at 82°C, while the mutant began to aggregate at 72°C.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com