Sentence examples for experimental models has shown from inspiring English sources

Data obtained from a host of experimental models has shown that low-intensity US can reversibly modulate the physiological activity of neurons in peripheral nerves, spinal cord, and intact brain circuits.

Autologous or allogeneic cell transplantation in experimental models has shown that these cells can engraft in the lung and differentiate into mature epithelial phenotypes [ 69, 70] and thus increase the cellular response to injury [ 71].

Experimental models have shown that axonal regeneration occurs across a supercharged end-to-side (SETS) nerve coaptation.

Additional work in experimental models have shown that Seprafilm had no effect on sepsis or abscess development when Seprafilm placement was concomitant with bacterial contamination [38] when placed in a surgical model of bowel injury [11] and that it did not exaggerate intra-abdominal septic conditions or induce a systematic inflammatory response [61].

Multiple investigations in various experimental models have shown that sepsis is associated with a decrease in capillary density in association with increased heterogeneous perfusion in visualized capillaries, such that capillaries with intermittent or no flow are found in close proximity to well-perfused capillaries [4 7].

Several experimental models have shown that overexpression of SR-BI in murine liver decreases the extent of atherosclerosis, even though it decreases HDL-plasma cholesterol concentration [22], [23], [24], [25].

Although there are no current data in humans, many experimental models have shown that NF-κB blockade (by different maneuvers, including IκB overexpression, NF-κB decoy oligonucleotides, NF-κB inhibitors, ACE inhibition, statins, glucocorticoids and antioxidants) attenuates renal inflammation [4], [18], [26].

Recent findings in experimental models have shown an additive or synergistic antitumor effects of LFP therapy.

Animal experimental models have shown interesting results in hypovolemic and septic shock and have lead to some interesting clinical studies.

Experimental models have shown that the continuous, progressive "wave front of necrosis" largely depends on the duration of ischaemia.

Experimental models have shown that FHIT full or haplo insufficiency confers an increased sensitivity to carcinogen exposure [ 47].