Sentence examples for experiment inhibited from inspiring English sources

Sema3D, an agonist for both np1 and np2 [26], inhibited tumor formation completely in one experiment (data not shown) and in another experiment inhibited strongly though not completely tumor development even though it was not as highly expressed as the other semaphorins (Fig. 3, E F).

Serum free media (used in previous research involving Western blots for 16 hours prior to starting experiment) inhibited GIRK1 protein expression, possibly due to lack of K+ ions or estrogen.

Experiments using both comparable activity/mg (amount of WT and P446L-GKRP used in each experiment inhibited 10 m U/ml GCK by the same extent at 5 m m glucose in the absence of the phosphate esters) (7) and equimolar concentrations (either 0, 50, 100 or 150 m m of both regulatory proteins used per assay) (9) were conducted.

In the context of this controlled experiment, inhibiting a saccade leads to goal acquisition (i.e., a monetary reward) and thus the enhanced activity in VS and putative FEF was adaptive.

An ouabain concentration of 50 nℳ used in subsequent experiments, inhibited active Rb+ uptake by 12.7±4.4% (n=4).

NFI siRNA knockdown experiments inhibited about 40% of SULT1A1 expression in ZR-75-1 cellsuggestinging other regulatory mechanism(s) exist.

The PHA680632 concentrations used in our colony formation experiments inhibited very little phospho-histone H3 on serine 10 (data not shown).

Peptide 242 268 eliminated most bacteria at 3 μM and 300 nM, and in separate experiments inhibited growth by 90% at 10 30 nM.

Rather, the behaviour suggests that the strong magnetic field used in these experiments inhibited polar auxin transport: this pattern of auxin distribution is the same as that observed after drug-induced inhibition of the transport with N-naftalamic acid (NPA) or in the eir1 mutant (note that the EIR1 gene encodes transmembrane proteins capable of mediating cellular auxin efflux) [ 47, 54].

Our results suggest the stress conditions tested in these experiments inhibit TOR signaling and this inhibition leads to the repression (either direct or indirect) of all three complexes.

In our experiments, CO effects were additive with LPS in activating p38 MAPK, but unlike the mouse experiments, inhibiting p38 MAPK did not block CO suppression of IL-1β.