Compared with nonsmokers in both pregnancies, women who were smokers in both pregnancies generally experienced increased risks of stillbirth in the second pregnancy.
Workers exposed to benzene have consistently experienced increased risks of hematopoietic disorders and leukemias (Hayes et al. 2000; Lan et al. 2004; Savitz and Andrews 1997).
In populations reporting a decrease, gradual incidence declines began as early as 1999 but accelerated in 2002 after the early and widely publicized termination of the Women's Health Initiative (WHI) estrogen/progestin arm, in which the experimental group experienced increased risks of breast cancer [ 11].
Factory workers experienced increased risk of cough, wheezing, breathlessness, nasal symptoms, skin symptoms and ever asthma compared to office workers.
Such individuals experienced increased risk for development of life threatening disorders such as high blood pressure, diabetes, cardiovascular disease and cancer.
Workers exposed to sensitizing chemicals, including phenol-formaldehyde resin, experienced increased risk of cough (3.43, 1.20 9.87) and nasal symptoms (3.07, 1.05 9.00).
Offspring with African-American (relative to white American) mothers experienced increased risk (medium-sized effect) for SSD in one birth cohort [ 63], but not the other [ 104].
Analyses by sex and restricted race/ethnicity showed that men (p = 0.004) and women (p = 0.009) from all races/ethnicities, and that non-Hispanic whites (p = 0.0002) experienced increased risk with increasing duration smoked (data not shown).
Workers exposed to glass microfibers experienced increased risk of cough (adjusted OR 2.04), wheezing (adjOR 2.20), breathlessness (adjOR 4.46), nasal (adjOR 2.13) and skin symptoms (adjOR 3.89) and ever asthma (adjOR 3.51), the risks of breathlessness (95%CI 1.68 11.86) and skin symptoms (1.70 8.90) remaining statistically significant after adjustment for confounders.
Although patients in subgroup G1 experienced increased risk of late recurrence, this subgroup was not considered as a desired model for this study, because subgroup G1 showed an unbalanced distribution of breast cancer molecular subtypes (Table 2), and the upregulated gene cluster C2 was not directly correlated with late recurrence (Table 3).
A preliminary analysis of this study reported median values of 18% and 23%, respectively, with IMRT, and patients with a rectal V70 >15% experienced increased risk of late rectal toxicity 5. Notably, current technology can be used to achieve sparing goals which are more stringent, especially when pelvic lymph nodes are not included in the radiation field.
