Sentence examples for expected cell death from inspiring English sources

As expected, cell death was not observed in roots treated with water, and extensive cell death was observed in roots treated with ClsA.

As expected, cell death in the telencephalon was less severe at earlier embryonic stages.

As expected, cell death incidence was significantly increased in the Src siRNA-transfected MDA-MB-231 cells compared with that in the control siRNA-transfected cells (Fig. 5f).

As expected, cell death was not induced by a mutant form of Puma lacking a functional BH3 domain and was inhibited by the pan-caspase inhibitor zVAD-fmk (not shown).

Although we expected that cell death would be enhanced in the absence of NF-κB due to its well established pro-survival activity, we found, to our surprise, that MEFs which lacked p50, RelA and c-Rel were more resistant to H2O2 induced cell death than wt cells (Fig. 2A).

As expected, autophagic cell death was not remarkable in SAS-R tumors after FR.

As expected, the cell death and proteasome regulator GO terms were only detected in genes up regulated in late lactation milk.

Therefore, one might have expected that cell death would be sharply elevated immediately after irradiation and that the process would be completed a short time thereafter.

As expected, apoptotic cell death induced by TNF- α was markedly increased in TAK1−/−MEF cells, whereas the apoptosis was significantly decreased in TAK1-overexpressed TAK1−/−MEF cells than in the case of mock transfectants.

We expected increased cell death in the lateral menisci (LM) compared to medial menisci (MM) following traumatic impaction based on the higher risk of LM tearing following this type of joint impaction [ 4].

As expected, increased cell death was observed in the 3T8 cells transfected with IκBαSR (3T8/IκBαSR) compared to the 3T8 cells transfected with bacterial GST as an negative control (3T8/Con) at 4 and 8 hours after TNF stimulation, consistent with published observations that NF-κB blockade renders cells sensitive to TNF-mediated apoptosis.