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Two-dimensional 1H,15N-NMR experiments exhibited signal loss upon complexation consistent with the formation of high-molecular-weight species.
The −60 sentinel was weak elsewhere whereas the −228 sentinel exhibited signal in the limb buds (Fig. 6B).
Each individual patient exhibited signal changes in those left and right hippocampal areas that reached significance in group statistics (Fig. 4).
A number of clusters with unknown functions were identified as coding sequences which exhibited signal peptides, such as clusters 11 and 126.
More importantly, a significant number of probes that went previously undetected (i.e., below a negative control threshold; [ 6]) in the control sample exhibited signal intensities above this threshold in DAP-modified samples.
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The one in the diagram exhibited signaling that makes defense more accurate.
In addition, the 1H NMR also exhibited signals due to one β-glucopyranosyl unit [δ 5.08 (d, J = 7.0 Hz, H-1")].
1H-NMR spectrum of compound 4 exhibited signals at 10.2, 11.4 attributed to 2NH groups and 13.1 ppm assigned to OH group which exchangeable with D2O.
Tumors identified positive for HGF/c-MET ligand-receptor complexes exhibited signals that were at least 2-fold greater than the isotype assay control signals (data not shown).
Tumors exhibited signals at least 2-fold greater than the isotype assay control were scored positive for HGF/c-MET complexes.
Of all the immune genes investigated, two (Sphinx1 and Dnr1) exhibited signals of positive selection.
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