Epilepsy is a chronic disorder in which excessive signaling of the cerebral neurons causes recurrent seizures.
Among the issues are long handover latency and excessive signaling [1].
Since they are non-linear devices, they cause excessive signal attenuation and noise at higher frequencies and voltages.
Even though the Toll-like receptor (TLR) pathway is integral to inflammatory defense mechanisms, its excessive signaling may be devastating.
S1 nuclease was then applied to remove the excessive signal probes, the single-stranded RNA and the mismatch RNA/DNA hybrids.
In HMIPv6-based network architecture, Mobile Nodes (MNs) generate excessive signalling overhead, where they move at borders of different Mobility Anchor Point (MAP) domains.
Excessive signaling by DDR1 and DDR2 has been linked to the progression of various human diseases, including fibrosis, atherosclerosis and cancer.
On the other hand, efficient use of network-wide cache space requires possible cooperation among caching routers without the use of excessive signaling burden.
These results indicate that the medial PFC is important for decision-making and that excessive signaling at D1 receptors may contribute to decision-making impairments.
Excessive signaling from the Wnt pathways has been implicated in the development of numerous types of cancers (MacDonald et al., 2009; von Maltzahn et al., 2012).
