Genome-wide single nucleotide polymorpism (SNP) data, for example, will allow for quantifying these associations, and provide improved spatial and temporal resolution to disentangle the relative role of Pleistocene refugia in generating genetic diversity.
Paradigms involving learned and unlearned behavior, for example, have been described for quantifying a variety of clinical signs of motor impairment including paretic gait disorders, tremor, and coordination deficits.
Figure 1 shows an illustrative example to outline our approach for quantifying biofilm formation ability of P. aeruginosa for a specific nutrient condition.
For example, quantifying a value for R0 can help understand the number of birds that would need to be vaccinated, how quickly a virus can spread through a flock, and if culling would be necessary.
These examples serve to illustrate basic properties of our general methodology for quantifying robustness.
For example, quantifying the results is difficult, as time-budget studies have noted that a given amount of time (for example, an hour) may have different values, depending on when it occurs within a day, a week, or a year.
Further studies, for example quantifying the amount of receptors by immunocytochemistry, will be required to resolve this issue.
For example, quantifying circulating adhesion molecules, proatherogenic substances, antifibrinolytics, evaluation of serum markers of inflammation, etc.
For example, quantifying how α-helices rearrange in reaction to weak and strong MH binding peptides, binding processes by TR, co-receptors and superantigens.
For example, quantifying the presence and completeness of orthologous core eukaryotic protein sequences (Parra et al. 2007) provides first intuition on the comprehensiveness of the assembly.
