Scientists are investigating, for example, defects in cellular mitochondria, which are responsible for producing energy.
For example, defects on reducible oxides help to stabilize atomically dispersed metal atoms on supports in the form of metal-O-support bonding [4].
Some of the defects in the structure of the nanotube can weaken a nanotube's strength, for example, defects in atomic vacancies or a rearrangement of the carbon bonds.
For example, defects in osteoblasts could contribute to delayed bone age seen in patients or reduced overall growth.
For example, defects in energy metabolism have been demonstrated in the skeletal muscle from patients with HD [53], [54].
For example, defects in dorsal hair regrowth in ankyrin-B+/− mice (Figure 4D) are indicative of impaired stem cell regeneration.
For example, defects in CTK1, CTF4, NUP133 and members of the THO complex all have been previously implicated in mediating repair responses to DNA damage [61] [63], [54].
For example, defects in maturation of the 40S subunit or changes in level of RP gene expression may be associated with bone marrow failure and cancer susceptibility syndromes in humans [16], [17].
A number of MOFs have received attention because of their photoluminescence properties and have been recently reviewed.[ 113]–[ 115] It is well known that defects play a considerable role in the optical properties of diverse solid-state materials.[ 114] For example, defect-induced luminescence has been found in Co2+-doped anatase, Cd-rich CdS and ZnS nanoparticles, and Eu2+-doped Sr2MgSi2O7.
For example, defects in Sr2MgSi2O7 act as electron traps close to the material's conduction band.
For example, defects of certain tRNA modifications result in decreased translation efficiency and increased translational error.
