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To shed light on the experimental conversion of a rigidly invariant code to a mutating code, the present study examined code mutations determining the propagation of Bacillus subtilis on Trp and 4-, 5- and 6-fluoro-tryptophans.
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Among alternatives examined, codes as simple as parity are shown to effectively correct single, self-identifying disk failures.
When sequence of the gene in several inbreds was examined, coding variations were noted, and abilities to utilize ferulic and p-coumaric acids differed.
Rather than examine each individual coding or noncoding region separately, we examined coding and noncoding DNA as concatenated sequences of individual regions.
We also examined coding potentials and protein sequences in the current study.
In primary care data we also examined codes indicating contact with secondary care.
We also examined coding haplotypes that were previously shown to affect PERK activation [ 31].
More importantly, among all the factors examined, coding sequence (CDS) length has the strongest (positive) correlation with protein evolutionary rates.
To identify the most salient deprecated codes, we examined codes in the Partners Healthcare Research Patient Data Registry (RPDR), an enterprise-wide research repository at Partners Healthcare.
For orthogonal evidence that popCons and popUncons elements were correctly classified as conserved or unconserved, respectively, we examined coding SNPs within genes found in these regions.
We also examined coding variants for genes at the chromosome 7 locus using the PROVEAN prediction tool (Choi and Chan, 2015).
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