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Hence, even though the differences in longevity that we observed across the three selection regimes was small, the fact that we observed them at all (with in 50 generations of selection) underscores the importance of sexual conflict in evolution of life span and aging.
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However, only few studies have so far addressed the correlation between sexual conflict and evolution of life-span and aging.
Sexual conflict has been suggested to have major consequences in the evolution of life-span and aging by affecting baseline investment in reproduction [ 16, 17].
The elucidation of the mechanisms that underlie the sexual antagonistic pleiotropy demonstrated in this study will be of great interest both to studies of life span determination and evolution of antagonistic functions within a single gene.
Of more importance, mutations that decrease the activity of the orthologs of these proteins in higher eukaryotes also extend life span, suggesting that the roles of these kinases in the regulation of life span are conserved along evolution [ 13- 17].
Voyager provides flexible life spans for agents, by supporting a variety of life span methods.
The limits of life span are finite.
Alternatively, extension of life span by rapamycin may require chico.
In contrast, high condition-dependent mortality led to the evolution of longer life span, supporting a key role of condition dependence in the evolution of aging.
We observed the evolution of shorter life span under high random mortality, confirming the classic prediction.
We present experimental evidence for the evolution of longer life span under high condition-dependent mortality.
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