Evidence of normalized auditory P50 suppression with acute nicotine in schizophrenia has supported the contention that elevated smoking rates in this disorder may be an attempt to correct a nicotinic receptor pathophysiology that may underly impaired sensory gating in these patients.
These findings provided the first direct evidence of how auditory cortical neurons alter their firing patterns during spontaneous vocalization and demonstrated that the predominant effects were suppressive in nature.
We also obtained evidence of enhanced auditory and ocular systems in the Chinese alligator.
Kossoff et al. [ 26] evaluated EEG findings and language function in six children with benign epilepsy with centrotemporal spikes (BECTS), and they also looked for additional evidence of impaired auditory comprehension and verbal memory.
We agree that the bulk of our data, from an auditory accumulation of evidence task, provides evidence only about accumulation of auditory evidence, and that we should correct our writing wherever it gives a misleading impression.
Other studies also reported difficulty to process speech in the presence of background noise, even with normal hearing [ 21], worse performance on dichotic listening compared to controls [ 22, 23], and evidences of cortical auditory dysfunction based on electrophysiological measures [ 24, 25].
