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There are several lines of evidence, discussed below, that suggest the implausibility of this proposed mechanism.
A number of recent lines of evidence, discussed below, suggest that WRN is potentially one such factor.
When combined with the corresponding neuropsychological evidence (discussed below), the results suggest that these tuning curves, even in occipital cortex, are likely to reflect true conceptual and not solely stimulus form distinctions.
Sedimentological evidence (discussed below) indicates the development of an increasingly arid climate throughout the course of Chinle deposition, probably driven by the movement of western North America out of the tropics and into the drier mid-latitudes (e.g., [87], [102]).
In the present study, we have gathered several lines of experimental evidence (discussed below) which definitively show that the formation of DNCB-specific Abs plays an important role in the development of DNCB-induced CHS.
In addition three pairs were excluded based on the phylogenetic evidence discussed below.
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In addition, there is also evidence from both clinical (discussed below) and experimental studies that, under specific circumstances, astrocytes or reactive astrocytes have the potential to exert a variety of other detrimental effects through gain of abnormal effects.
Secondary contact could also result in a bimodal distribution of phenotype, but cichlid ecology, a single colonization event and no genetic evidence for secondary admixture (discussed below), suggests this is not the case in Apoyeque.
The evidence from these studies (discussed below) suggests that NK cells can affect the development of autoimmunity through several mechanisms, including suppressing viral infections and potential subsequent autoimmune responses, modulating autoreactive responses of other immune cells, or, as effector cells, directly mediating tissue damage (Fig. 2).
Indeed, as discussed below, different evidence suggests to bring HC into the TACs group [3].
As discussed below, considerable evidence indicates that another important function of VIP and PACAP is to modulate the activity of inflammatory cells.
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